Встановлено достовірний вплив типу вищої нервової діяльності на вміст загального протеїну (F = 11,2 > FU = 2,90; Р < 0,001), альбумінів (F = 14,6 > FU = 2,90; Р < 0,001), глобулінів (F = 3,02 > FU = 2,90; Р < 0,05), β-глобулінів (F = 4,07 > FU = 2,90; Р < 0,05), γ-глобулінів (F = 5,52 > FU = 2,90; Р < 0,01), лактату (F = 36,2 > FU = 2,90; Р < 0,001) та пірувату (F = 4,1 > FU = 2,90; Р < 0,01), загального холестеролу (F = 4,1 > FU = 2,90; Р < 0,05) та триацилгліцеролів (F = 4,9 > FU = 2,90; Р < 0,01) в крові собак. Тоді, як впливу вищої нервової діяльності на вміст глюкози (F = 0,69 < FU = 2,90; р = 0,56) та α-глобулінів (F = 1,50 < FU = 2,90; р = 0,24) в плазмі крові собак не встановлено. Короткотермінова харчова депривація також достовірно впливала на вміст глюкози (F = 18,7 > FU = 4,15; Р < 0,001), β-глобулінів (F = 39,3 > FU = 4,15; Р < 0,001), γ-глобулінів (F = 11,7 > FU = 4,15; Р < 0,001), альбумінів (F = 19,1 > FU = 4,15; Р < 0,001), глобулінів (F = 7,71 > FU = 4,15; Р < 0,01), лактату (F = 50,0 < FU = 4,15; Р < 0,001), пірувату (F = 20,7 < FU = 4,15; Р < 0,001), загального холестеролу (F = 5,7 < FU = 4,15; Р < 0,05) та триацилгліцеролів (F = 11,5 < FU = 4,15; Р < 0,01) в крові собак. Впливу харчової депривації на вміст загального протеїну (F = 0,44 < FU = 4,15; р = 0,28) та α-глобулінів (F = 0,88 < FU = 4,15; р = 0,36) в крові піддослідних собак не встановлено. Протягом усього експерименту міжфакторної взаємодії між короткотерміновою харчовою депривацією і типом вищої нервової діяльності не встановлено (F = 0,01–1,29 < FU = 2,90; р = 0,29–1,00), що вказує на відсутність впливу депривації на тип вищої нервової діяльності тварин
собаки, вища нервова діяльність, депривація, метаболізм, білки, жири, вуглеводи
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